antibody anti nr2b rabbit polyclonal proteintech (Proteintech)
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Proteintech
antibody anti nr2b rabbit polyclonal proteintech
Antibody Anti Nr2b Rabbit Polyclonal Proteintech, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 174 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rabbit+polyclonal+anti+nr2b/NMDAR2B%2FGRIN2B+Antibody/10__7554_slash_elife__90116-265-69-74
Average 96 stars, based on 174 article reviews
Antibody Anti Nr2b Rabbit Polyclonal Proteintech, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 174 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rabbit+polyclonal+anti+nr2b/NMDAR2B%2FGRIN2B+Antibody/10__7554_slash_elife__90116-265-69-74
Average 96 stars, based on 174 article reviews
antibody anti nr2b rabbit polyclonal proteintech - by Bioz Stars,
2026-10
96/100 stars
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Western Blot:Article Title: TMEM25 modulates neuronal excitability and NMDA receptor subunit NR2B degradation Article Snippet: .. The primary antibodies and dilutions used for the Western blot were as follows: rabbit polyclonal anti-TMEM25 (Proteintech, catalog 24361-1-AP), Article Title: TMEM25 modulates neuronal excitability and NMDA receptor subunit NR2B degradation Article Snippet: .. The primary antibodies and dilutions used for the Western blot were as follows: rabbit polyclonal anti-TMEM25 (Proteintech, catalog 24361-1-AP), Blocking Assay:Article Title: TMEM25 modulates neuronal excitability and NMDA receptor subunit NR2B degradation Article Snippet: .. Membranes were blocked with blocking reagent (Beyotime Biotechnology, catalog P0252) for 15 minutes and then incubated overnight at 4°C with rabbit polyclonal anti-TMEM25 (Proteintech, catalog 24361-1-AP), Article Title: TMEM25 modulates neuronal excitability and NMDA receptor subunit NR2B degradation Article Snippet: .. Membranes were blocked with blocking reagent (Beyotime Biotechnology, catalog P0252) for 15 minutes and then incubated overnight at 4°C with rabbit polyclonal anti-TMEM25 (Proteintech, catalog 24361-1-AP), Incubation:Article Title: TMEM25 modulates neuronal excitability and NMDA receptor subunit NR2B degradation Article Snippet: .. Membranes were blocked with blocking reagent (Beyotime Biotechnology, catalog P0252) for 15 minutes and then incubated overnight at 4°C with rabbit polyclonal anti-TMEM25 (Proteintech, catalog 24361-1-AP), Article Title: TMEM25 modulates neuronal excitability and NMDA receptor subunit NR2B degradation Article Snippet: .. Membranes were blocked with blocking reagent (Beyotime Biotechnology, catalog P0252) for 15 minutes and then incubated overnight at 4°C with rabbit polyclonal anti-TMEM25 (Proteintech, catalog 24361-1-AP), Article Title: Resveratrol Improves Synaptic Plasticity in Hypoxic-Ischemic Brain Injury in Neonatal Mice via Alleviating SIRT1/NF-κB Signaling-Mediated Neuroinflammation. Article Snippet: Neonatal hypoxic-ischemic encephalopathy (HIE) is an obstinate disease that troubles neonatologists.. At present, cognitive impairment after HIE has received increasing attention.. Synaptic plasticity determines the development of cognitive function, so it is urgent to develop new drugs that can improve HIE-induced cognitive impairment. Saline:Article Title: TMEM25 modulates neuronal excitability and NMDA receptor subunit NR2B degradation Article Snippet: .. Membranes were blocked with blocking reagent (Beyotime Biotechnology, catalog P0252) for 15 minutes and then incubated overnight at 4°C with rabbit polyclonal anti-TMEM25 (Proteintech, catalog 24361-1-AP), Article Title: TMEM25 modulates neuronal excitability and NMDA receptor subunit NR2B degradation Article Snippet: .. Membranes were blocked with blocking reagent (Beyotime Biotechnology, catalog P0252) for 15 minutes and then incubated overnight at 4°C with rabbit polyclonal anti-TMEM25 (Proteintech, catalog 24361-1-AP), other:Article Title: Protective effect of 3-n-butylphthalide against intrastriatal injection of malonic acid-induced neurotoxicity and biochemical alteration in rats. Article Snippet: The rabbit polyclonal antibodies against NR2B, PSD95, Akt, GSK3β, β-catenin and the mouse monoclonal antibodies against ARC, Molecular Weight:Article Title: Resveratrol Improves Synaptic Plasticity in Hypoxic-Ischemic Brain Injury in Neonatal Mice via Alleviating SIRT1/NF-κB Signaling-Mediated Neuroinflammation. Article Snippet: Neonatal hypoxic-ischemic encephalopathy (HIE) is an obstinate disease that troubles neonatologists.. At present, cognitive impairment after HIE has received increasing attention.. Synaptic plasticity determines the development of cognitive function, so it is urgent to develop new drugs that can improve HIE-induced cognitive impairment. Electrophoresis:Article Title: Resveratrol Improves Synaptic Plasticity in Hypoxic-Ischemic Brain Injury in Neonatal Mice via Alleviating SIRT1/NF-κB Signaling-Mediated Neuroinflammation. Article Snippet: Neonatal hypoxic-ischemic encephalopathy (HIE) is an obstinate disease that troubles neonatologists.. At present, cognitive impairment after HIE has received increasing attention.. Synaptic plasticity determines the development of cognitive function, so it is urgent to develop new drugs that can improve HIE-induced cognitive impairment. SDS Page:Article Title: Resveratrol Improves Synaptic Plasticity in Hypoxic-Ischemic Brain Injury in Neonatal Mice via Alleviating SIRT1/NF-κB Signaling-Mediated Neuroinflammation. Article Snippet: Neonatal hypoxic-ischemic encephalopathy (HIE) is an obstinate disease that troubles neonatologists.. At present, cognitive impairment after HIE has received increasing attention.. Synaptic plasticity determines the development of cognitive function, so it is urgent to develop new drugs that can improve HIE-induced cognitive impairment. |
![Atomoxetine induced changes on Grin2B messenger (m)RNA and <t>NR2B</t> levels. Notes: Male adolescent rats were treated for 21 days (postnatal days 21–41) with atomoxetine hydrochloride (3 mg/kg, intraperitoneal [ip]) or saline (0.9%, ip). The striatum (STR), mesencephalon (MES), and hippocampus (HC) of the early treatment group were analyzed 24 hours after the last ip application, whereas the brains of rats assigned to the late treatment group were probed after a treatment-free period of 2 months. ( A ) Glutamate receptor ionotropic N-methyl-D-aspartate 2B (Grin2B) expression was probed in the STR, MES, and HC. Bar diagrams depict mean values of quantitative reverse transcription polymerase chain reaction (qRT-PCR) measurement. ( B ) Bar diagrams depict mean values obtained by densitometric quantification of the ( C ) respective Western blots detecting the glutamate receptor ionotropic N -methyl- D -aspartate 2B (NR2B) at a molecular weight of ∼170 kDa. Controls were male age-matched saline-treated rats. All data are presented as ± standard error of the mean; early treatment group n=7; late treatment group n=8; * P <0.05; ** P <0.01.](https://pub-med-central-images-cdn.bioz.com/pub_med_central_ids_ending_with_7115/pmc03857115/pmc03857115__dddt-7-1433Fig5.jpg)